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JOSEPH F. MAHER, M.D.
Associate Professor
Program Director
Medical Genetics Residency Program
Phone: 214-648-1600
Mailing Address:
5323 Harry Hines Blvd, MC 8591
Dallas, Texas 75390-8591
E-mail: joseph.maher@utsouthwestern.edu
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Research Interest:
- Medical genetics
- Control of gene expression and cell-fate determination
- Cell signaling in human genetic disorders
Lab Personnel
Education:
- Bachelor of Science (Zoology), magna cum laude, University of Texas - Austin, TX 1982
- Doctor of Medicine, University of Texas Southwestern Medical School - Dallas, TX 1987
Licensure / Certifications:
- Diplomat of the American Board of Internal Medicine, 9/90
- Diplomat of the American Board of Medical Genetics (Clinical Genetics), 9/96
Professional Positions:
- 2007 - Present - Associate Professor, McDermott Center for Human Growth and Development, UT Southwestern
- 1995 - 2006 - Associate Professor, Department of Medicine, Division of Hematology, University of Mississippi Medical Center and Medical Staff / Research Service (151) G.V. (Sonny) Montgomery Veterans Affairs Medical Center
- 1992 - 1995 - Associate, Howard Hughes Medical Institute, Department of Molecular Biology and Genetics / Lab of Dr. Daniel Nathans, Johns Hopkins University School of Medicine
- 1991 - 1995- Fellowship in Medical Genetics, Department of Medicine and Center for Medical Genetics, Johns Hopkins University School of Medicine - Baltimore, MD
- 1987 - 1990 -Internship/Residency in Internal Medicine - University of Texas Southwestern Medical School, Parkland Memorial Hospital and Dallas Veterans Affairs Medical Center
Recent Publications:
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Wilson, J.G., Hoff Lindquist, J., Grambow, S.C., Crook, E.D., and Maher, J.F. Potential Role of Increased Iron Stores in Diabetes. Am. J. Med. Sci. 325: 332-339 (2003).
- Ventura-Holman, T. , Lu, D., Si, X., Izevbigie, E.B., and Maher, J.F. The Fem1c Genes: Conserved Members of the Fem1 Gene Family in Vertebrates. Gene 314: 133-139 (2003).
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Lu, D, Ventura-Holman, T., Li, J., McMurray, R.W., Subauste, J.S. & Maher, J.F. Abnormal Glucose Homeostasis and Pancreatic Islet Function in Mice with Inactivation of the Fem1b Gene. Molecular & Cellular Biology 25: 6570-6577 (2005).
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Ventura-Holman, T., Hahn, H., Subauste, J.S., and Maher, J.F. The Fem1a gene is down-regulated in Rhabdomyosarcoma. Tumor Biology 26: 294-299 (2005).
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Maher, J.F.; Hines, R.S.; Futterweit, W.; Crawford, S.; Lu, D.; Shen, P.; Oefner, P.; Kazi, M.; Wilson, J.G.; Subauste, J.S.; & Cowan, B.D. FEM1A is a Candidate Gene for Polycystic Ovary Syndrome. Gynecol Endocrinol 21: 330-335 (2005).
For additional publications: Search PubMed
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