McDaniel, LD, R. Prueitt, L.C. Probst, K.S. Wilson, D. Tomkins, G.N. Wilson and R.A. Schultz, "Novel assay for Roberts Syndrome assigns variable phenotypes to one complementation group." Am. J. Med. Genetics., 93(3):223-229, 2000
McDaniel, L.D., N. Chester, M. Watson, A.D. Borowsky, P. Leder, R.A. Schultz., "Allele-Specific Modulation of chromosome instability in mouse models for Bloom Syndrome." DNA Repair, 2(12):1387-1404, 2003
Nahari, D., L.D. McDaniel, L.B. Task, R.L. Daniel, S. Velasco-Miguel, E.C. Friedberg., "Mutations in the Trp53 Gene of UV-Irradiated Xpc Mutant Mice Suggest A Novel Xpc-Dependent DNA Repair Process." DNA Repair, 1(3):379-386, 2004
Theron T., M.I. Fousteri, M. Volker, L.W. Harries, E. Botta, M. Stefanini, M. Fujimoto, J.O. Andressoo, J. Mitchell, N.G, Jaspers, L.D. McDaniel, L.H. Mullenders, A.R. Lehmann, "Transcription-associated breaks in xeroderma pigmentosum group D cells from patients with combined features of xeroderma pigmentosum and Cockayne syndrome." Molecular & Cellular Biology, 25(18):8368-8378, 2005
Paul C. Porter, P.C., D.R. Clark, L.D. McDaniel, W.G. McGregor, J.C. States, "Telomerase-immortalized human fibroblasts retain UV-induced mutagenesis and p53-mediated DNA damage responses" DNA Repair, 5(1):61-70, 2006
Fischhaber, P.L., L.D. McDaniel, E.C. Friedberg, "DNA Polymerases for Translesion DNA Synthesis: Enzyme Purification and Mouse Models for Studying Their Function" Methods in Enzymology, In Press 2006
SIGNIFICANT PUBLICATIONS
McDaniel, L.D. and R.A. Schultz, "Elevated sister chromatid exchange phenotype of Bloom Syndrome cells is complemented by human chromosome 15." Proc Natl Adac Sci. USA., 89:7968-7972, 1992
Luo, G., Santoro, I., McDaniel, L.D., Nishijima, I., Mills, M., Youssoufian, H., Vogel, H., Schultz, R.A., Bradley, A., "Cancer predisposition caused by elevated mitotic recombination in Bloom Mice." Nature Genetics, 26(4):424-429, 2000
McDaniel, L.D., D.J. Tomkins, E.J. Stanbridge, M.J. Somerville, E.C. Friedberg, R.A. Schultz, "Mapping of a single locus capable of complementing the defective heterochromatin phenotype of roberts syndrome cells" Am J Hum Genet., 77(1):132-139, 2005
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